and the conversion is as I said as above as far as I knew.  Hope that this helps.  The levels as it states are half way down I think...

PRECOCIOUS PUBERTY

Precocious puberty is defined as the appearance of puberty before the age of 8 in girls. The condition can be divided into GnRH dependent central precocious puberty (CPP) and GnRH independent pseudoprecocious puberty. The rise of serum LH after GnRH administration in CPP is indistinguishable from the normal pubertal rise. If extrapituitary secretion of gonadotropins, or sex steroid secretion independent of pituitary gonadotropins, leads to signs of puberty, the condition is incomplete precocity or pseudoprecocious puberty.

Premature pubarche. The condition is caused by premature activation of adrenal androgen secretion. The GnRH response is prepubertal, and dehydroepiandrosterone (DHEA) and dehydroepiandrosterone sulfate (DHEA-S) concentrations appropriate for pubic hair stage (Table 1). The half-life of DHEA is relatively short, with large diurnal and pulsatile variations in serum concentrations. On the contrary, the half-life of DHEA-S is long, and its measurement is therefore clinically more meaningful except in short stimulating tests. If premature pubarche is associated with strongly advanced growth and bone age or signs of virilization, a detailed endocrine examination is needed. In congenital virilizing adrenal hyperplasia androgen secretion is strongly increased, both DHEA as well as testosterone and androstenedione. The serum 17-hydroxyprogesterone concentration is very high, in untreated cases usually more than 50 nmol/L and often several hundreds of nmol/L, and is therefore a useful diagnostic indicator. A dexamethasone test (1 mg/m² for 7 days) should be made. In congenital adrenal hyperplasia the increased hormonal levels are normalized but in cases of adrenal tumor they may not. Virilizing adrenal tumors are often malignant and should immediately be diagnosed and removed. In cases of tumors the growth curve is bent.

In late-onset forms of congenital adrenal hyperplasia the symptoms are more modest and clinical onset later. The frequency of these conditions show large international differences The regional prevalence need to be known for planning a reasonable workup. A serum sample taken in the morning, in the follicular phase of cycling women, is useful as screening in low incidence populations. A serum 17-hydroxyprogesterone value below 2 nmol/L makes 21-hydroxylase deficiency unlikely, and a level above 6 nmol/L likely. If the likelihood of a defect is considered high, or the basal value is ambiguous, a iv ACTH test is performed with sampling at 0 and 30 or 60 minutes for serum 17-hydroxyprogesterone.

Late onset 3beta-hydroxysteroid dehydrogenase is more rare. It can be suspected based on high DHEA or DHEA-S levels, and confirmed by ACTH-stimulated serum 17-hydroxypregnenolone higher than 10 SD above normal mean values, and elevated 17-hydroxypregnenolone/17-hydroxyprogesterone ratio (Sakkal-Alkaddour et al., 1996).

Other forms of pseudoprecocious puberty. Some rare cases of pseudoprecocious puberty can be due to gonadotropin-producing tumors. Ovarian cysts, granulosa or theca cell tumors are the cause of precocious puberty in about 10 % of the cases. In the McCune-Albright syndrome, polyostotic fibrous dysplasia and café-au-lait spots are associated with precocious puberty. The new assays are helpful in distinguishing this GnRH independent condition with suppressed serum FSH concentrations below the normal prepubertal range from GnRH dependent precocious puberty (Apter and Sipilä, 1993).

True precocious puberty. In true precocious puberty, gonadotropin and gonadal steroid concentrations and the response to GnRH are generally in the normal pubertal range. As RIA is unable to measure low LH concentrations, the standard procedure to diagnose Gn-dependent precocious puberty has been the GnRH test. A stimulated concentration of LH above 7-10 IU/L indicates central precocious puberty. A single sample at 20-40 minutes is enough. A rapid, subcutaneous, single-sample gonadotropin-releasing hormone (GnRH 100 microg) stimulation test was found indistinguishable from intravenous GnRH stimulation test in the evaluation of CPP (Eckert et al., 1996). Random LH levels in excess of 0.3 IU/L had 100% specificity for CPP (Neely et al., 1995b).