that in pregnancy of a woman with normal functioning adrenal glands, carrying a CAH baby, the Mothers adrenals would in a sense pick up the high testosterone and to some extent possibly be raising the levels of cortisol naturally to try and combat this.  It is all about negative feedback and that is possibkly the difference there as to why a woman with normal adrenal glands STILL carries that baby to full term.  It would be because in a sense, her adrenals are just working harder.  However, in the situation whereby a CAH woman is pregnant and not treated adequately, not only is the baby exposed to that, but the woman is making higher levels of testoserone further into the pregnancy.  Thats because the pituritary would sense this and secrete ACTH in response in an attempt to recitfy this siutation.  However, this Mother cannot possibly meet that demand and more testosterone would be made. 

I have NO clue about 3 beta CAH.  However, I do have a feeling that somehow the situation is different than when I had Ashley.  Like I say my body could make the adequate amounts of cortisol to balance things.  To my mind if Bebe had high testoserone levels and a history of pre term small babies (premature) then somewhere, her needs are not being met here where hormaone balances are concerned.  It stands to reason as the pregnancy advances the situation of high testosterone is going to elevate much higher if you consider what cortisol normally does during pregnancy.

THAT is the way my mind has been assessing things for the most part here.