Sorry Karen - yes I do have CAH (not LOCAH), so perhaps my answer was inappropriate - sometimes I do forget where I am, lol! I like the LOCAH board as very often it is adults here. Also the manifestation of LOCAH fascinates me...but I digress...
I do think that in the past when LOCAH people were on higher doses of steroids that possibly there were more adrenal crisis incidents than there are now, since doctors are generally prescribing lower and more realistic doses.
It is a fact that when corticosteroid doses are above a certain dosage (and it varies for each of the corticosteroids) that ANYONE on longterm steroids is more susceptible to adrenal crisis during stress and illness - not just CAH people. This occurs, as you know because the adrenal gland stops producing its own supply of cortisol when it is being supplied by another source eg. via tablets (HPA axis and all that).
What steroid dose are you on Karen? Also do you know what mutations you have? There are significant variations in LOCAH symptoms depending often on the mutations that you have. If you have two LOCAH mutations then you are less likely to have additional problems than if you have say a deletion (total absence of function) and a LOCAH mutation (such as V281L). In this case having eg. a deletion and LOCAH mutation you will still be classified as a LOCAH but the lack of function on the other more severe allele appears to influence the severity of symptoms. Perhaps this may be a clue to your conundrum!
